Share Success: Letters From Readers
I: From Toddi N.
Submit Form on Wednesday, January 09, 2013 at 22:29:19
After a year on EZorb, my DEXA T score of my left femoral hip has gone from -3.1 to -2.8, a 10% change.
I'm grateful and now focusing on what MORE I can do to improve my numbers. The plan is diet, EZorb continuance and now I am working with a personal trainer to lift weights. I will keep you posted.
II: From Melvin F.
Submit Form on Friday, November 16, 2012 at 11:00:47
Hi, my name is Melvin Freedenberg and I am an EZorb user for more than three years, now. I ordered it for eight bone spurs in my fingers, all of which disappeared in five months after starting an EZorb regimen.
For about ten years, I have suffered from numbness in both hands, first in my pinkies and finally progressing to all parts of both hands. The problem only occurred when I slept and the numbness constantly woke me up.
In March of 2009 I had an MRI done on my upper spine, near my neck. The MRI report said that I had moderate osteoarthritis in C5-6 and C6-7. The nerves to my arms and hands eminate from those parts of my spine. Eureka!
In order to get around the collapse of my spine during sleep, I bought a cervical collar which helped a great deal. I continued to take EZorb in the hope that it would arrest, if not reverse, my osteoarthritis. I have always used a Tempurpedic mattress and pillow and sleep on my side. After more than three years of taking EZorb, I now sleep like a baby and four months ago, I gave up the cervical collar because I didn't need it anymore.
For those of you who don't get immediate results, don't give up. Eventually, EZorb will do everything it is supposed to do and more.
III: From Janice C.
Submit Form on
Sunday, October 14, 2012 at 17:26:07
Hi, my name is Janice. I was recently diagnosed with heel spurs, torn cartilage in wrist. I already have reflux sympathetic dystrophy in my right foot, osteoarthritis in lower back and right knee.
I haven't been able to kneel on my knees in three years. I take one step at a time because of the pain. When I would get up from a sitting position to walk I took baby steps cause of the pain. I could barely walk. I didn't want surgery so I started to search for other remedies.
I first tried the heel cups no help. Then I found this amazing product called ezorb I took double the dose cause I have so much wrong and in three months I am almost pain free.
Much more than that I have a zip to my walk again. I have been telling everyone about your product. But more important they see what it has done for me.
Thank you so very much,
IV: From Shawn B., Oklahoma City, OK, USA
Submit Form on
Wednesday, September 05, 2012 at 13:49:40
Me and my family have been taking ezorb. It has been a miracle.
My wife have fibromyalgia and heel spurs. It has almost stopped her pain.
My son has bilateral knees. He was having knee pain, requiring pain meds around the clock. He has not taken any pain meds since 2-3 weeks after starting ezorb. He is scheduled to have a MRI in November. I am so excited to see the results.
His physician wanted to put him on Fosamax. But after looking at the side effects. My wife and I decided to just try ezorb.
the Desk of EZorb Newsletter Editor:
newsletter is now read by over 70,000 subscribers
worldwide. Success stories you have contributed over
the years have had a great impact on many people's
quality of life. Your continuous support will be
greatly appreciated by tens of thousands who have been
suffering and would continuously suffer, without your
help! Please email your
story to sharesuccess @ elixirindustry.com
or simply post it at Testimonial
Submit Form. Your personal information will never be
revealed to the public.
Research News: Salt Restriction Has Multiple Vascular Benefits
Restricting salt intake reverses vascular endothelial dysfunction in people with moderately increased blood pressure (BP), shows a randomized study published in the Journal of the American College of Cardiology.
The effects were independent of changes in the participants' systolic BP, leading the researchers to suggest that salt causes vascular dysfunction beyond its effect on BP.
Editorialists David Celermajer (Royal Prince Alfred Hospital, Sydney, Australia) and Bruce Neal (Sydney Medical School) sound a note of caution, however, saying that simply controlling for BP is unreliable because of "enormous moment-to-moment within-individual variability" of BP.
But they say that the study is a reminder that "a simple molecule such as sodium can assault our vessels in complex maladaptive ways," adding: "Our next challenge is to translate this knowledge derived from vascular biology into effective and appropriate public health strategy."
The study had a crossover design. The 17 participants reduced their dietary sodium intake to a target of about 50 mmol/day, verified by urinary excretion and dietary analysis. For one 5-week period, the participants took a daily slow-release sodium chloride tablet that brought their salt intake up to about 150 mmol/day. For the other 5-week period of the study they took a placebo tablet.
Salt restriction had notable effects on both macrovascular and microvascular function, report study author Douglas Seals (University of Colorado at Boulder, USA) and team.
Flow-mediated dilation of the brachial artery was 68% greater during restricted than usual salt intake, at 6.01% versus 3.57%. Flow-mediated dilatation increased during restricted salt intake in all but two participants.
There were similar benefits for the microcirculation, with forearm blood flow increased by 42% in response to intrabrachial artery infusion of acetylcholine during salt restriction and BP-corrected forearm vascular conductance increased by 52%. Again, the effects were seen in all but two participants.
The team found that the endothelial benefits of salt restriction were mediated by increased bioavailability of nitric oxide and tetrahydrobiopterin, and by reduced oxidative stress; infusion of antioxidants improved endothelial function during normal but not restricted salt intake.
As anticipated, systolic BP was significantly lower during restricted- versus usual-salt intake, at an average of 128 versus 140 mmHg.
Celermajer and Neal note that "normal" salt intake as defined by current standards is far higher than what was consumed for most of human evolution. "Likewise, although a generation of clinicians has learned that 'normal' systolic blood pressure is approximately 100 mmHg plus age, it is now evident that this is not the physiological norm either."
Original research was published in J Am Coll Cardiol 2013; 61: 335–343, 344–345.
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